Schizophrenia

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Kitchen Remedies and Natural Cures for Schizophrenia
1200 mg of desiccated thyroid daily for 100 days

Kitogenic Diet

Eliminate Wheat, Sugar, and Known Allergens (Safe Harbor claims it can take up to five year, but 90% success).

vitamin: B3, B6, multiple B, Vitamin C, zinc, and manganese

High doses of B3, B6, and zinc (Dr Hoffer)

supplements: selenium, cysteine, glutamine, and tryptophan

calcium and selenium

treat for lime disease

eliminate stress

wear red to lower cortisol levels

fasting (Soviets found this works) They would throw the person into a locked room and leave him there for a couple of days. Kitogenic Diet would have been nicer.

lowered serotonin is the root to the “Jekyll and Hyde” personality disorder (Merk Manual, home ed. 1997)

fructose malabsorption See: Getting Started Saccharine Disese: http://www.journeytoforever.org/farm_library/Cleave/cleave_toc.html and fructose malabsorption http://en.wikipedia.org/wiki/Hydrogen_breath_test

Food Grade Diatomaceous Earth See Parasites

Schizophrenia: Sub-category of Psychotic Mental Disorders
Schizophrenia is a group of symptoms, not an individual illness. One must understand that it has a medical and legal definition. Up until the 1980’s doctors relied on the broad definition as codified in the ICD-9 (International Statistical Classification of Diseases). The ICD began using classifications established by Dr. Bertillon in 1893. The ending digit notates revisions. ICD-6, in 1949, added in a mental disorder section. In 1977, the World Health Organization made ICD-9 public domain. The National Center for Health Statistics used the ICD-9 to create the ICD-9-CM. It likewise is being constantly revised. ICD-10-CM is currently being revised by ICD-11-CM. Likewise, he World Health Organization, through ICD-10 and ICD-11, continues to revise those definitions. The American Psychiatric Association (APA) created their own system, the Diagnostic and Statistical Manual of Mental Disorders (DSM), and parallels the ICD classifications. DSM Section V outlines the accepted criteria for schizophrenia. Insurance, government agencies, and other related services rely on these codes to determine eligibility for services and compensation. These codes are convenient for doctors, insurance companies, and government agencies, but does little to improve accurate  diagnosis or treatment for the patients. Schizophrenia is sub-divided into five sub-categories: Paranoid, disorganized, catatonic, and undifferentiated. If the patient is under 15 years old, it is then termed Early Onset Schizophrenia. The child could present a wide variety of symptoms and conditions such as delusions, hallucinations, disorganized speech or behavior, and/or catatonic behavior. Since it is a category and not a specific illness in itself, there is no set of definitive causes. Studies conflict as to whether it is genetic, environmental, or possibly both. Well into the 1980’s, most doctors lumped autism, bipolar, multiple personality, and all childhood psychosis together as early onset schizophrenia. Instead of being a true diagnosis, childhood schizophrenia was just a catchall phrase. Many doctors and studies attempted to differentiate symptoms and criteria but no agreement persisted. For example, Rutter 1974, based on the fact that autistic children do not develop delusions or hallucinations, urged that autism get its own separate classification. In essence, doctors realized that they needed to re-define, subdivide, and differentiate between childhood schizophrenia, psychosis, and other similar disorders. Simply put, schizophrenia is difficult to diagnose because what is acceptable at one age is not acceptable another age. A very small child may have an imaginary friend and be considered normal. But a teen who has an imaginary friend would be considered abnormal. According to schizophrenia.com, there can be ten years between the first symptoms and the diagnosis. It appears once everything else is ruled out, then the doctor finally gives the diagnoses of schizophrenia.

Schizophrenia Research
Currently, research suggests that childhood schizophrenia may be physical. MRIs can detect fluid filled cavities in the middle of the brain. (Rapoport Archives of General Psychiatry October.) There also seems to be some abnormality in the temporal lobe. Elkis 1995. One study claims that older and younger parents have offspring with higher incidents of social disorders. Parents between ages 25-29 had the best chance of having normal children, but it did not say that parental age had any definitive influence on who becomes schizophrenic. (Schizophrenia bulletin vol 34 6pp 1042-1046) 2008. In a previous issue, The Schizophrenia Bulletin (Vol33 issue 4, pp 912-920) says that a study needs to show that a particular situation is high for schizophrenia and not for other similar disorders in order to infer a connection or precursor. For this reason, the parents' age cannot be considered a factor for schizophrenia, despite being a predictor for social disorders. Genetics and environmental trigger may combine to create a higher incident of schizophrenia. The general population has a 1% chance of developing schizophrenia whereas those with a parent or sibling have a 10% chance. That means 10 times more as likely to get schizophrenia. Yet 60% of schizophrenics have no relatives with the disorder. Some studies think stress, which produces higher levels of cortisol, is a trigger if the person is predisposed. Other studies link low levels of dopamine and glutamate. In reality, none of the theories or studies found any true links. Identical twin who share identical genes have shown that heredity cannot be the cause despite higher percentages run in family as opposed to family without any schizophrenia. So a gene might make one more inclined to get it but is not definitive. Industrialization accompanied higher rates of schizophrenia. That would be environmental and not genetics. Ireland appears to have a 16 times the world average. Studies seem to indicate that thyroid and brain atrophy. One study by Danziger claims that 1200 mg of desiccated thyroid daily for 100 days will cure schizophrenia. Some studies say that supplements selenium, cysteine, glutamine, and tryptophan produce good results. If early onset schizophrenia is genetic, then one would reason that a set of twins, who share the exact same genetics, would both get schizophrenia, but that only rarely happens. Contrary to reason, the twin argument does NOT disprove genetic inheritance. In the former Soviet Republic (1950), a set of twins, conjoined from the lower intestines and downward, Masha and Dasha, (in addition to an identical gene pool) shared circulatory, digestive, excretory, lymphatic, hormonal, and lymphatic systems, but had two distinct spines. Despite having the same germs in their systems, at the same time, they did not get flus, colds, or other illnesses at the same time. Masha and Dasha prove that inherited genetics are not manifested identically in identical twins. The fact that identical twin do not both get schizophrenia does not rule out genetic predisposition. Some evidence suggests physical triggers. Early onset schizophrenia patients tend to have damaged thyroid glands and/or swollen brain ventricles. The incidence of early onset schizophrenia increased dramatically along with industrialization. Early onset schizophrenia is more prevalent in developed countries than undeveloped countries. Torrey, E.F., and Miller, J. (2002). The invisible plague: The rise of mental illness from 1750 to the present. New Brunswick: Rutgers University Press. Schizophrenics tend to be born during the coldest time of the year, to poor families who live just outside the business districts, and ate food low in calcium and selenium. Torrey, E.F. (1987). Prevalence studies in schizophrenia. British Journal of Psychiatry, 150, 598-608. One researcher, Brown, found a link between ticks carrying lime disease and a higher incident of early onset schizophrenia. Brown, J.S. Jr. (1994). Geographic correlation of schizophrenia to ticks and tick-borne encephalitis. Schizophrenia Bulletin 20(4), 755-775.

Any animal can cross infect humans. One cat parasite, toxoplasma gondiican, causes brain Cancer in humans. There is evidence that the parasite (toxoplasma gondiican) also causes Schizophrenia, dillusions, and psychotic disorders. If Schizophrenia can be caused by parasites, then that is a course of action. See Parasites.

Sugar malabsorption. Results from a study: "A higher national dietary intake of refined sugar and dairy products predicted a worse 2-year outcome of schizophrenia. A high national prevalence of depression was predicted by a low dietary intake of fish and seafood." 

Fructose malabsorption See: Getting Started Saccharine Disese: http://www.journeytoforever.org/farm_library/Cleave/cleave_toc.html and fructose malabsorption http://en.wikipedia.org/wiki/Hydrogen_breath_test

Also see the great plains laboratory

Schizophrenia Holistic Methods
There are holistic approaches where patients claim improvement. Some claim that desiccated liver improved their symptoms. Hoffer, A. (2001). Thyroid and schizophrenia. Journal of Orthomolecular Medicine, 16(4), 205-212. One article by “Safe Harbor” claims that 90% of schizophrenia can be cure by eliminating all refined sugar (and products containing refined sugar), elimination of any known allergens, and supplementation of Vitamins B3, B6, a multiple B, Vitamin C, zinc, and manganese. Some patients may take five years or more on the diet to recover. Furthermore, Safe Harbor claims that none of their patients' symptoms worsened while on the diet. Safe Harbor (2002). Alternative Mental Health On-line. The standard recommended “schizophrenia” nutritional and dietary regimen: A remarkable 90% recovery rate. http://www.alternativemental health.com/articles/schizdiet.htm

Rid the body of parasites. Does the parasite or the increased stress cause the illness? Impossible to know, but clearing the body of parasites does improve the patient. See Parasites for common remedies. For some people, it is a cure. I would take Food Grade Diatomaceous Earth, even for a remote chance to end mental illness or emotional disturbances. The former Soviet Union had no private pharmaceutical companies to promote pill popping. Their cure did not involve any drugs. They found that fasting cured psychotic episodes. When a patient was having a full blown psychotic episode, the doctors quit feeding the patient and the outburst ended almost instantaneously. Cott, A. (1971). Controlled fasting treatment of schizophrenia. Schizophrenia, 3, 2-10. Fasting might be likened to the kitogenic diet used in America to cure epilepsy. http://en.wikipedia.org/wiki/Ketogenic_diet ” Several years ago Dr. Hoffer, a pioneer in nutritional treatments, found that higher doses of vitamin B3 could help schizophrenics. He found that acute symptoms of schizophrenia correlated with a certain substance, known as the mauve factor, which is detectable in urine. The chemical in the urine was named the mauve factor because it would change the test paper to this color. Original treatments with B3 evolved into further successes with B6 and zinc. The mauve factor is now known as hydroxyhemo pyrrolin-2-one (HPL for short). Further studies suggest that HPL may be elevated in other neurological disorders such as autism, Down syndrome, depression, ADHD and drug addictions [Source: McGinnis]. Research and testing for the mauve factor continued in small scientific circles, spanning the last thirty to forty years, but was often only in relationship to neurological complaints. They discovered that the mauve factor is likewise related to patients with chronic fatigue, arthritis, irritable bowel, heart disease and even cancer.” http://health.howstuffworks.com/diseases-conditions/chronic-fatigue/the-mauve-factor.htm These findings build a strong case that environmental factors, such as diet, may be the trigger. The diet remedies appear to work better than the prescription regiments. The hypothesis that dopamine was a factor has been proven false, instead adrenochrome has been proven to be the triggering substance. When epinephrine is oxidized it creates the pigment adrenochrome. Hoffer, A. (1982). The adrenochrome hypothesis of schizophrenia revisited. Journal of Orthomolecular Psychiatry, 10(2), 1-21. Stress can also be a trigger because it also produces high levels of adrenochrome, which can cause hallucinations and paranoia. Dishinger, R.C. (1998). Bad behavior and illness are caused by biochemical imbalances. Owensboro, KT: Medici Music Press. Certain genes also play a role in creating schizophrenia such as the catechol-O-methyltransferase gene or the GSTM1*0 allele. These two genes overly produce adrenaline and adrenochrome when the patients is under stress, encounters an allergen, or eats sugar; this results in psychotic episodes. Adolescents and young adults already have rising hormone levels. Therefore when they sugar binge or great are under great stress, the adrenaline rises enough to elevate adrenochrome, resulting in a psychotic outburst. {Hoffer, A. (1992). The adrenochrome hypothesis of schizophrenia revisited. Journal of Orthomolecular Psychiatry, 10(2), 1-21. Berkow, R., Beers, M.H., Bogin, R.M., and Fletcher, A.J. (eds). (1997). The Merk Manual of Medical Information Home Edition. New York: Pocket Books.}

Adrenochrome creates a domino effect. It damages the thyroid, lowers serotonin, tryptophan, niacin, glutathione peroxidase, superoxide dismutase, and catalase. All of this creates a lowered defense system. The lowered serotonin is the root to the “Jekyll and Hyde” personality disorder. The schizophrenic patient does not have to have a defective gene. Pathogens encountered during gestation also can predispose the person to schizophrenia. If the fetus had high levels of adrenochrome during gestation, it will have a higher risk for schizophrenia. Brown, J.S. Jr. (1994). Geographic correlation of schizophrenia to ticks and tick-borne encephalitis. Schizophrenia Bulletin, 20(4), 755-775. Buka, S.L., Tsuang, M.T., Torrey, E.F., Klebanoff, M.A., Bernstein, D., and Yolken, R.H. (2001). Maternal infections and subsequent psychosis among offspring. Archives of General Psychiatry, 58(11), 1032-1037.

Common pharmaceuticals only help up to 25% of schizophrenic patients. Horrobin, D.F. (2002). Evidence-based medicine and the need for noncommercial clinical research directed towards therapeutic innovation. Experimental Biology and Medicine, 227(7), 435-437.

That leaves 75% of the schizophrenic patients helpless. Conversely, diet, supplementation, and fasting have up to and past 90% improvement. Some claim it is a cure. Why aren't the pharmaceutical companies promoting holistic medicine? The reason is that they cannot patent or make large profits on food and supplements. So doctors are shamed away from supplements and toward traditional drugs. Yet the adrenochrome hypothesis shows a higher success rate than the pharmaceuticals who promote glutamate or dopamine.

This connection was known as early as 1955. ( Journal of Mental Science (1955) 101:538-547. doi: 10.1192/bjp.101.424.538 Adrenochrom as the Cause of Schizophrenia: Investigation of Some Deductions from this Hypothesis. A.J. Lea, MB ChB)